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Старый 24.12.2017, 02:35   #28
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Protein Restriction
Toward the goal of getting more sodium out of the body, out of damaged cells,
Gerson eliminated not only sodium from the diet; he also eliminated protein from the diet
for a period of time. In his experiments, as Dr. Ward noted, Gerson had extraordinary
laboratory support in the best equipped medical and scientific community in the world at
that time. He was able to observe that once you put somebody on a high-potassium, lowsodium
diet, the first thing that happens is that tremendous quantities of sodium are
excreted in the urine.
Where does it come from? It’s coming from inside individual damaged cells. In a
really sick person, with extensive tissue damage syndrome, tissues all over the body are
dumping sodium. Because sick people got better when they dumped sodium in the urine,
Gerson wanted to increase that effect and prolong it. He found that by eliminating dietary
protein, he could cause even more of what he called “Natrium Ausschuss”, sodium
outpouring, or sodium flooding out in the urine – more, and more, and more.
The problem with extreme protein restriction is that you can’t do it for too long,
because then you being to compromise immunity. This has been observed for a long time.
Science has long known that protein is necessary for good immunity, but has never known
how much. It has been assumed, wrongly, that we should have lots of it, and that we
should always have it.
Gerson, however, said the opposite. He said you must stop dietary proteins for a
period of six to eight weeks in order to cause sodium to leave damaged cells and in order to
cause edema to be absorbed. In his mind, it was clear that sodium was trapped in the body
with protein; it was trapped in deposits of protein and sodium that were somehow
complexed together. This is accurate. It is accurate within the context of Ling’s work, and
Ling’s work is modern-day biophysics.
We know now, from the work of Robert Good, that protein restriction, something
that you’re doing on the Gerson Therapy, can actually stimulate cell-mediated immunity. Tlymphocyte
activity can be stimulated by protein restriction.
Robert Good was the Director of the Sloan-Kettering Institute for Cancer Research.
Prior to his position with Sloan-Kettering, Good spent time in Egypt visiting a friend in
Alexandria who had been working with malnourished children. Good took a deep interest in
the immune profiles of these long-malnourished children. He asked his friend why certain
panels of the immune profile were disturbed, why they were off, and his friend said, “We
don’t know. We just know that they are, but we don’t know which dietary deficiency is
causing which immune abnormality”. Good decided it was high time to do some basic
research to answer some of these questions.
When he arrived at Sloan-Kettering, he set up a guinea pig experiment, a very
simple experiment. He had laboratory chow that contained no protein. He took this noprotein
lab chow and fed it to group A, and group B was given normal chow. Group A
received no protein. Group B was the control group, the putatively well-fed guinea pig. Good
had expected to see deterioration of serum and cell-mediated immunity. Serum immunity is
antibody production, key to some of our bacterial and viral immunity, the ability to fight
some bacteria and viruses. Cell-mediated immunity is conducted by T-cells – lymphocytes –
and these are the ones that fight bacteria, fungi, and also fight tumors. Good predicted
failure of, at least, serum immunity. He was unprepared for what he saw. Not only did
serum immunity remain stable, but lymphocytes, especially T-lymphocytes – the thymus
lymphocytes – became tremendously increased nonspecifically active, and remained
aggressively and nonspecifically active for a long period of time.
And at that point, Good realized and wrote that he had stimulated immunity by dietary
restriction of protein. This led to a long series of experiments in many laboratories, all
related to Robert Good, who is known as the most published pathologist in the western
medical literature. His experiments have shown, in one animal model after another,
diseases which are called long term or degenerative diseases – often genetically
predetermined – in mice, guinea pigs, and other animals, can be affected by protein and
calorie restriction. Some of these diseases have been direct analogues of human diseases,
and the weight of the evidence strongly suggests similar effects in man.
Calorie restriction is another aspect of your treatment here. How can that be when
you’re eating all the time? Because the fats are gone from the diet. A tablespoon of
carbohydrate and a tablespoon of protein yield approximately the same number of calories.
A tablespoon of fat provides more than double that number of calories. Fats are everywhere
in the Western diet, in our civilized diet; bakery goods, cakes, candies, rolls, meats,
cheeses, fried foods, nuts, and seeds. But not in this diet. In this diet the only fats are those
in oatmeal, which is 1.5% its total calories in fat – that’s why it congeals when it gets cool –
and individual fatty acids through some of the vegetables and fruits – individual and small
number of them, I might add – and, of course, the flax oil. The patient receives about
ninety calories a day in fats.
What we mean, when we say we have a protein-calorie restricted diet here is that we
have a better diet. We don’t keep people off of supplemental protein for too long. Six to
eight weeks is all we can do without compromising immunity to some extent. However, it is
entirely safe as we use it, because we give nonfat dairy proteins after six to eight weeks.
This provides plenty of protein for the patient.
In this dietary program, even as patients receive it in the early weeks, there is
enough protein input from the highly bio-available protein content of potatoes, oatmeal,
vegetables and vegetable juices to offset daily obligatory protein loss. A typical patient loses
about 40 grams of protein a day through entrails – obligatory protein loss – but that is
mostly replaced through the basic vegan diet already before adding the dairy protein. When
you add the dairy protein, you will have 30-40 grams more than you require. You’re kept in
what’s called positive nitrogen balance.
Good and his coworkers established that protein and calorie restriction can do some
really quite remarkable things with animal models. The first mouse that was studied
extensively was the (NZB X NZW) F1 (B/W) mouse, called NZB for short. This mouse is a
very rare direct analog mouse. The disease it develops, systemic lupus erythematous, is a
direct human analog. That means that it is the same disease in the mouse and the human,
and if you can affect it in the mouse, you can affect it in the human.
The NZB mouse, when protein-calorie restriction is implemented, will not develop
lupus. This is a mouse genetically preprogrammed to develop lupus. Protein-calorie
restriction initiated at weaning will prevent the development of an otherwise inevitable
disease. Even if the disease is allowed to develop, it can be caused to regress by initiating
protein-calorie restriction after the disease has presented.
Another mouse, the kdkd mouse, gets vascular lesions and has a tendency toward
nephropyosis. These mice, if protein-calorie restriction is initiated at weaning will not
develop blood vessel lesions, and plaque, and kidney problems. Kidney problems can
develop when blood vessel supplies are pinched off. The same is true with the heart. You
cut off the blood supply and organs get into trouble, and muscles get into trouble. Kdkd
mice, even if they are allowed to develop the disease, can be regressed if protein-calorie
restriction is initiated after the disease presents.
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